A REVIEW OF อ่านเพิ่มเติม

Keap1 also reversibly translocates to the p62-gels in a very p62-binding dependent style to activate the transcription component Nrf2. Mice deficient for Atg8-conversation-dependent selective autophagy exhibit that impaired turnover of p62-gels leads to Nrf2 hyperactivation in vivo. These success reveal that p62-gels will not be simple substrates f